A. Tardivel

Discipline

Histoire, Philosophie et Sociologie des sciences (2) Linguistique (1)

Type de document

Articles (35)

Co-auteur

Date

Source

Mot-clé

Mice (30) famille (22) Families (22) Animals (22) Family (22) Human beings (19) Receptors (19) protéine (17) Tumor necrosis factor (14) nécrose (12) Necrosis (12) Cells (12) Necrosis (12) Development (11) spectacle (10) ligne (10) Amino acid sequence (10) membrane (9) Proteins (9) B cells (9) gène (8) Ligands (8) Drama (8) survie (7) Drama (7) Survival (7) Ectodermal dysplasia (7) Ability (7) Phenotype (6) Teeth (6)

Documents écrit par A. Tardivel (35) | parle de A. Tardivel (0)

LRR-protein RNH1 dampens the inflammasome activation and is associated with COVID-19 severity.
G. Bombaci et al.
A loop region of BAFF controls B cell survival and regulates recognition by different inhibitors.
M. Vigolo et al. (23 mars 2018)
Ribonuclease inhibitor 1 regulates erythropoiesis by controlling GATA1 translation.
V. Chennupati et al. (2018)
Ectodysplasin A in Biological Fluids and Diagnosis of Ectodermal Dysplasia.
J. Podzus et al. (2017)
Antibodies That Block or Activate Mouse B Cell Activating Factor of the Tumor Necrosis Factor (TNF) Family (BAFF), Respectively, Induce B Cell Depletion or B Cell Hyperplasia.
C. Kowalczyk-Quintas et al. (16 sept. 2016)
AIM2 inflammasome is activated by pharmacological disruption of nuclear envelope integrity.
A. Di Micco et al. (9 août 2016)
T Cell Priming by Activated Nlrc5-Deficient Dendritic Cells Is Unaffected despite Partially Reduced MHC Class I Levels.
G. Rota et al. (2016)
NLRP3 Inflammasome Is Expressed and Functional in Mouse Brain Microglia but Not in Astrocytes.
A. Gustin et al. (2015)
Cutting edge: IL-1α is a crucial danger signal triggering acute myocardial inflammation during myocardial infarction.
J. Lugrin et al. (2015)
Epithelial NAIPs protect against colonic tumorigenesis.
R. Allam et al. (2015)
Stoichiometry of heteromeric BAFF and APRIL cytokines dictates their receptor binding and signaling properties.
S. Schuepbach-Mallepell et al. (2015)
Generation and characterization of function-blocking anti-ectodysplasin A (EDA) monoclonal antibodies that induce ectodermal dysplasia.
C. Kowalczyk-Quintas et al. (2014)
The Nlrp3 inflammasome regulates acute graft-versus-host disease.
D. Jankovic et al. (2013)
No Evidence That Soluble TACI Induces Signalling via Membrane-Expressed BAFF and APRIL in Myeloid Cells.
J. Nys et al. (2013)
Recognition of gut microbiota by NOD2 is essential for the homeostasis of intestinal intraepithelial lymphocytes.
W. Jiang et al. (2013)
ER stress activates the NLRP3 inflammasome via an UPR-independent pathway.
P. Menu et al. (2012)
Molecular and therapeutic characterization of anti-ectodysplasin A receptor (EDAR) agonist monoclonal antibodies.
C. Kowalczyk et al. (2011)
Atherosclerosis in ApoE-deficient mice progresses independently of the NLRP3 inflammasome.
P. Menu et al. (2011)
Rescue of the mature B cell compartment in BAFF-deficient mice by treatment with recombinant Fc-BAFF.
L.K. Swee et al. (2010)
Functional analysis of Ectodysplasin-A mutations causing selective tooth agenesis.
G. Mues et al. (2010)
Biological activity of ectodysplasin A is conditioned by its collagen and heparan sulfate proteoglycan-binding domains.
L.K. Swee et al.
Malarial hemozoin is a Nalp3 inflammasome activating danger signal.
C. Dostert et al. (2009)
TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts.
C. Bossen et al. (2008)
Intestinal bacteria condition dendritic cells to promote IgA production.
J.C. Massacand et al. (2008)
Significant correction of disease after postnatal administration of recombinant ectodysplasin A in canine X-linked ectodermal dysplasia.
M.L. Casal et al. (2007)

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