J. Loffing

Discipline

Education (1) Biological anthropology (1)

Type

Articles (12)

Co-author

Date

Source

Keyword

Expression (12) Sodium (11) Mice (11) protein (9) Diet (9) dietary habit (9) Animals (8) Control (7) Kidney tubules (7) Aldosterone (7) plasma (7) standard (5) loss (5) gene (5) Balances (Weighing instruments) (5) Transportation (5) Salt (5) Equilibrium (5) Blood pressure (5) transport (5) salt (4) adult (4) Phenotype (4) Scarcity (4) Phosphorylation (4) Homeostasis (4) Excretion (4) Excretion (4) adult (4) well (3)

Documents written by J. Loffing (12) | is about J. Loffing (0)

The serine-threonine kinase PIM3 is an aldosterone-regulated protein in the distal nephron.
A. Spirli et al.
Dietary sodium induces a redistribution of the tubular metabolic workload.
K. Udwan et al. (Nov 15, 2017)
Severe hyperkalemia is rescued by low-potassium diet in renal βENaC-deficient mice.
E. Boscardin et al.
Renal tubular SGK1 deficiency causes impaired K+ excretion via loss of regulation of NEDD4-2/WNK1 and ENaC.
D. Basquin et al. (Aug 1, 2016)
Severe Salt-Losing Syndrome and Hyperkalemia Induced by Adult Nephron-Specific Knockout of the Epithelial Sodium Channel α-Subunit.
R. Perrier et al.
Adult nephron-specific MR-deficient mice develop a severe renal PHA-1 phenotype.
J. Canonica et al.
Extracellular K(+) rapidly controls NaCl cotransporter phosphorylation in the native distal convoluted tubule by Cl(-) -dependent and independent mechanisms.
D. Penton et al. (2016)
ENaC activity in collecting ducts modulates NCC in cirrhotic mice.
D. Mordasini et al. (2015)
Mice carrying ubiquitin-specific protease 2 (Usp2) gene inactivation maintain normal sodium balance and blood pressure.
D. Pouly et al. (2013)
Common noncoding UMOD gene variants induce salt-sensitive hypertension and kidney damage by increasing uromodulin expression.
M. Trudu et al. (2013)
Renal tubular NEDD4-2 deficiency causes NCC-mediated salt-dependent hypertension.
C. Ronzaud et al. (2013)
alphaENaC-mediated lithium absorption promotes nephrogenic diabetes insipidus.
B.M. Christensen et al. (2011)