B. Thorens

Discipline

Education (4) History, Philosophy and Sociology of Sciences (3) Linguistics (2) Management (2) Sociology (1) Economies and finances (1) Archaeology and Prehistory (1)

Type

Articles (69) Reviews (5)

Co-author

Date

Source

Keyword

Mice (55) Glucose (44) Animals (34) Insulin (33) Diabetes (33) Expression (30) Secretion (29) Secretion (29) Control (26) Human beings (25) Non-insulin-dependent diabetes (23) analysis (22) Genes (22) Analysis (22) Cells (18) plasma (18) Islands (17) Homeostasis (17) gene (16) show (15) Toleration (14) Glucagon (14) Development (13) Hypothalamus (13) stress (12) Neurons (12) Liver (12) Hypoglycemia (12) tolerance (12) approach (11)

Documents written by B. Thorens (69) | is about B. Thorens (0)

Neuronal glucose sensing mechanisms and circuits in the control of insulin and glucagon secretion.
B. Thorens (Oct 1, 2024)
Correction: Cold-induced dishabituation in rodents exposed to recurrent hypoglycaemia.
K. Vickneson et al.
Correction: Chronic hyperglycaemia increases the vulnerability of the hippocampus to oxidative damage induced during post-hypoglycaemic hyperglycaemia in a mouse model of chemically induced type 1 diabetes.
A.D. McNeilly et al.
GLP-1 metabolite GLP-1(9-36) is a systemic inhibitor of mouse and human pancreatic islet glucagon secretion.
N.R. Gandasi et al.
Disrupted hypothalamic transcriptomics and proteomics in a mouse model of type 2 diabetes exposed to recurrent hypoglycaemia.
F. Marzetta et al.
Correction to: Disrupted hypothalamic transcriptomics and proteomics in a mouse model of type 2 diabetes exposed to recurrent hypoglycaemia.
F. Marzetta et al.
The glucose transporter 2 regulates CD8+ T cell function via environment sensing.
H. Fu et al.
Hyperglycemia increases SCO-spondin and Wnt5a secretion into the cerebrospinal fluid to regulate ependymal cell beating and glucose sensing.
F. Nualart et al.
Tmem117 in AVP neurons regulates the counterregulatory response to hypoglycemia.
S. Gaspari et al. (Aug 3, 2023)
Chronic hyperglycaemia increases the vulnerability of the hippocampus to oxidative damage induced during post-hypoglycaemic hyperglycaemia in a mouse model of chemically induced type 1 diabetes.
A.D. McNeilly et al.
Identification of biomarkers for glycaemic deterioration in type 2 diabetes.
R.C. Slieker et al. (May 3, 2023)
Oral metformin transiently lowers post-prandial glucose response by reducing the apical expression of sodium-glucose co-transporter 1 in enterocytes.
L. Zubiaga et al. (Apr 21, 2023)
A caveolin-1 dependent glucose-6-phosphatase trafficking contributes to hepatic glucose production.
J. Chilloux et al.
In vivo and in vitro characterization of GL0034, a novel long-acting glucagon-like peptide-1 receptor agonist.
B. Jones et al.
GLUT2 expression by glial fibrillary acidic protein-positive tanycytes is required for promoting feeding-response to fasting.
M.J. Barahona et al. (Oct 21, 2022)
Structural and molecular characterization of paraventricular thalamic glucokinase-expressing neuronal circuits in the mouse.
S. Gaspari et al.
Hypothalamic Irak4 is a genetically controlled regulator of hypoglycemia-induced glucagon secretion.
A. Picard et al.
Homocysteine Metabolism Pathway Is Involved in the Control of Glucose Homeostasis: A Cystathionine Beta Synthase Deficiency Study in Mouse.
K. Meneyrol et al. (May 25, 2022)
Opposite physiological and pathological mTORC1-mediated roles of the CB1 receptor in regulating renal tubular function.
L. Hinden et al. (Apr 4, 2022)
Neuronal regulation of glucagon secretion and gluconeogenesis.
B. Thorens
Plasma triacylglycerols are biomarkers of β-cell function in mice and humans.
C. Roujeau et al.
Fgf15 Neurons of the Dorsomedial Hypothalamus Control Glucagon Secretion and Hepatic Gluconeogenesis.
A. Picard et al.
Persistent or Transient Human β Cell Dysfunction Induced by Metabolic Stress: Specific Signatures and Shared Gene Expression with Type 2 Diabetes.
L. Marselli et al. (Dec 1, 2020)
Hypoglycemia-Sensing Neurons of the Ventromedial Hypothalamus Require AMPK-Induced Txn2 Expression but Are Dispensable for Physiological Counterregulation.
S. Quenneville et al.
The Constitutive Lack of α7 Nicotinic Receptor Leads to Metabolic Disorders in Mouse.
B. Gausserès et al. (Jul 16, 2020)

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