H. Abriel

Discipline

Sciences de l'information et de la communication (2) Psychologie (1)

Type de document

Articles (19) Recensions (4)

Co-auteur

Date

Source

Mot-clé

Mice (10) Expression (10) Cells (10) Sodium channels (9) Sodium (8) puits (7) Human beings (7) Example (7) modèle (6) Channels (6) Heart (6) Arrhythmia (6) gène (5) Methods (5) Muscle cells (5) Animals (5) Methods (5) Brugada syndrome (5) protéine (4) mutation (4) effet (4) densité (4) Density (4) Genes (4) Ubiquitin (4) Heart cells (4) Will (4) Experiments (4) Electrocardiography (4) Drugs (4)

Documents écrit par H. Abriel (19) | parle de H. Abriel (0)

Multiomics uncover the proinflammatory role of Trpm4 deletion after myocardial infarction in mice.
M. Boukenna et al. (1 avr. 2023)
A Distinct Pool of Nav1.5 Channels at the Lateral Membrane of Murine Ventricular Cardiomyocytes.
J.S. Rougier et al. (2019)
Transcriptomic analyses of murine ventricular cardiomyocytes.
M. Chevalier et al. (2018)
Post-translational modifications of voltage-gated sodium channels in chronic pain syndromes.
C.J. Laedermann et al. (2015)
β1- and β3- voltage-gated sodium channel subunits modulate cell surface expression and glycosylation of Nav1.7 in HEK293 cells.
C.J. Laedermann et al. (2013)
Rufinamide Attenuates Mechanical Allodynia in a Model of Neuropathic Pain in the Mouse and Stabilizes Voltage-gated Sodium Channel Inactivated State.
M.R. Suter et al. (2013)
Mice carrying ubiquitin-specific protease 2 (Usp2) gene inactivation maintain normal sodium balance and blood pressure.
D. Pouly et al. (2013)
Dysregulation of voltage-gated sodium channels by ubiquitin ligase NEDD4-2 in neuropathic pain.
C.J. Laedermann et al. (2013)
Mapping genetic variants associated with beta-adrenergic responses in inbred mice.
M. Hersch et al. (2012)
Cardiovascular response to beta-adrenergic blockade or activation in 23 inbred mouse strains.
C. Berthonneche et al. (12 août 2009)
Molecular autopsy in sudden cardiac death and its implication for families: discussion of the practical, legal and ethical aspects of the multidisciplinary collaboration.
K. Michaud et al. (2009)
Mineralocorticoid receptor is essential for corticosteroid-induced up-regulation of L-type calcium currents in cultured neonatal cardiomyocytes.
J.S. Rougier et al. (2008)
Analyses of a novel SCN5A mutation (C1850S): conduction vs. repolarization disorder hypotheses in the Brugada syndrome.
S. Petitprez et al. (2008)
Roles and regulation of the cardiac sodium channel Na v 1.5: recent insights from experimental studies.
H. Abriel (2007)
The KCNQ1 potassium channel is down-regulated by ubiquitylating enzymes of the Nedd4/Nedd4-like family.
T. Jespersen et al. (2007)
Propagation velocity kinetics and repolarization alternans in a sheep model of pacing-induced atrial fibrillation.
Y. Prudat et al. (2006)
Brugada syndrome and fever: genetic and molecular characterization of patients carrying SCN5A mutations.
D.I. Keller et al. (2005)
Molecular and clinical determinants of drug-induced long QT syndrome: an iatrogenic channelopathy.
H. Abriel et al. (2004)
Channel openings are necessary but not sufficient for use-dependent block of cardiac Na(+) channels by flecainide: evidence from the analysis of disease-linked mutations.
H. Liu et al. (2002)