K. Bouzourene

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Education (3)

Type

Articles (10)

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Mice (8) model (5) disease (5) Apolipoprotein E (5) Inflammation (5) Expression (5) Example (5) Animals (5) disease (5) well (4) training (4) Training (4) Phenotype (4) Genes (4) Messenger RNA (4) Exercise (4) Diseases--Animal models (4) Atherosclerosis (4) Imaging (4) Exercise (4) Training (4) show (3) Development (3) Apolipoproteins (3) Week (3) Prison treadmills (3) Running (3) Muscles (3) Macrophages (3) Glucose (3)

Documents written by K. Bouzourene (10) | is about K. Bouzourene (0)

Physical Performance and Skeletal Muscle Transcriptional Adaptations Are Not Impacted by Exercise Training Frequency in Mice with Lower Extremity Peripheral Artery Disease.
J. Lavier et al. (Apr 16, 2023)
High-intensity exercise in hypoxia improves endothelial function via increased nitric oxide bioavailability in C57BL/6 mice.
J. Lavier et al.
Impact of aerobic exercise type on blood flow, muscle energy metabolism, and mitochondrial biogenesis in experimental lower extremity artery disease.
M. Pellegrin et al. (Aug 20, 2020)
Towards Quantification of Inflammation in Atherosclerotic Plaque in the Clinic - Characterization and Optimization of Fluorine-19 MRI in Mice at 3 T.
E. Darçot et al. (Nov 25, 2019)
Reactive oxygen/nitrogen species contribute substantially to the antileukemia effect of APO866, a NAD lowering agent.
A.J. Cloux et al. (Nov 19, 2019)
Experimental Drainage Device to Reduce Lymphoedema in a Rat Model.
V. Triacca et al.
Activation of Bone Marrow-Derived Cells Angiotensin (Ang) II Type 1 Receptor by Ang II Promotes Atherosclerotic Plaque Vulnerability.
M. Pellegrin et al. (Sep 4, 2018)
Voluntary Exercise Stabilizes Established Angiotensin II-Dependent Atherosclerosis in Mice through Systemic Anti-Inflammatory Effects.
M. Pellegrin et al. (2015)
Experimental peripheral arterial disease: new insights into muscle glucose uptake, macrophage, and T-cell polarization during early and late stages.
M. Pellegrin et al. (2014)
Atherosclerosis in ApoE-deficient mice progresses independently of the NLRP3 inflammasome.
P. Menu et al. (2011)