B. Desvergne

Discipline

Psychologie (2) Education (2) Science politique (1) Histoire, Philosophie et Sociologie des sciences (1)

Type de document

Articles (33) Recensions (5)

Co-auteur

Date

Source

Mot-clé

Mice (22) Animals (16) Receptors (14) Expression (13) gène (11) Example (11) spectacle (10) Metabolism (10) Cells (10) métabolisme (9) Genes (9) Transcription (9) Inflammation (9) Homeostasis (9) Transcription (9) Metabolism (9) modèle (8) Human beings (8) puits (7) effet (7) Development (7) Deletion (7) lipide (6) ADN (6) différenciation (6) règlement (6) Differentiation (6) Treatment (6) Transcription factors (6) Fat cells (6)

Documents écrit par B. Desvergne (33) | parle de B. Desvergne (0)

Representations of personalised medicine in family medicine: a qualitative analysis.
M.S. Boyer et al. (1 mars 2022)
Médecine personnalisée et prévention des maladies chroniques - Besoins, perceptions et attentes des patients et des médecins généralistes [Personalized medicine and chronic disease prevention: Needs, attitudes and expectations regarding in general practice]
C. Cohidon et al. (10 nov. 2021)
Sex Dimorphism of Nonalcoholic Fatty Liver Disease (NAFLD) in Pparg-Null Mice.
M. Schiffrin et al. (15 sept. 2021)
May direct-to-consumer genetic testing have an impact on general practitioners' daily practice? a cross-sectional study of patients' intentions towards this approach.
C. Cohidon et al. (26 avr. 2021)
DBnorm as an R package for the comparison and selection of appropriate statistical methods for batch effect correction in metabolomic studies.
N. Bararpour et al. (11 mars 2021)
Lack of Adiponectin Drives Hyperosteoclastogenesis in Lipoatrophic Mice.
M.B. Madel et al. (2021)
Anti-adipogenic signals at the onset of obesity-related inflammation in white adipose tissue.
T. Caputo et al.
Visualization and normalization of drift effect across batches in metabolome-wide association studies
N. Bararpour et al. (22 janv. 2020)
Systemic PPARγ deletion in mice provokes lipoatrophy, organomegaly, severe type 2 diabetes and metabolic inflexibility.
C. Winkler et al.
Renal mineralocorticoid receptor expression is reduced in lipoatrophy.
B. Toffoli et al.
System analysis of cross-talk between nuclear receptors reveals an opposite regulation of the cell cycle by LXR and FXR in human HepaRG liver cells.
L. Wigger et al. (2019)
Lack of Adipocytes Alters Hematopoiesis in Lipodystrophic Mice.
A. Wilson et al. (2018)
PPARγ Controls Ectopic Adipogenesis and Cross-Talks with Myogenesis During Skeletal Muscle Regeneration.
G. Dammone et al. (2018)
HDAC3 is a molecular brake of the metabolic switch supporting white adipose tissue browning.
A. Ferrari et al. (2017)
Nephropathy in Pparg-null mice highlights PPARγ systemic activities in metabolism and in the immune system.
B. Toffoli et al. (2017)
Integrative and systemic approaches for evaluating PPARβ/δ (PPARD) function.
G.M. Giordano Attianese et al. (2015)
Impaired musculoskeletal response to age and exercise in PPARβ(-/-) diabetic mice.
H. Fu et al. (2014)
Genome-wide analysis of SREBP1 activity around the clock reveals its combined dependency on nutrient and circadian signals.
F. Gilardi et al. (2014)
Morbillivirus glycoprotein expression induces ER stress, alters Ca2+ homeostasis and results in the release of vasostatin.
J.M. Brunner et al. (2012)
Genome-wide RNA polymerase II profiles and RNA accumulation reveal kinetics of transcription and associated epigenetic changes during diurnal cycles.
G. Le Martelot et al. (2012)
NCoR1 is a conserved physiological modulator of muscle mass and oxidative function.
H. Yamamoto et al. (2011)
The PPARbeta/delta agonist GW0742 relaxes pulmonary vessels and limits right heart hypertrophy in rats with hypoxia-induced pulmonary hypertension.
L.S. Harrington et al. (2010)
The pollutant diethylhexyl phthalate regulates hepatic energy metabolism via species-specific PPARalpha-dependent mechanisms.
J.N. Feige et al. (2010)
Peroxisome Proliferator-Activated Receptor beta/delta in the Brain: Facts and Hypothesis.
M.G. Hall et al. (2008)
Glycogen synthase 2 is a novel target gene of peroxisome proliferator-activated receptors.
S. Mandard et al. (2007)

next