B. Rückert

Type de document

Co-auteur

C.A. Akdis

O.F. Wirz

A. Wallimann

D. Verschoor

M. van Splunter

W. van de Veen

G. Tan

A. Straumann

B. Stanić

H. Spits

C.B. Schmidt-Weber

C. Rhyner

N. Ouaked

U. Ochsner

P.Y. Mantel

B.N. Lambrecht

H. Kuipers

T. Kubo

C. Karagiannidis

K. Jansen

D. Ignatova

D. Huntjens

M. Herrmann

R.W. Hendriks

E. Guenova

A. Globinska

R.J. Fonseca Guevara

L. Flatz

C. Fassnacht

R. Crameri

Y.T. Chang

F. Castro-Giner

O. Boyman

K. Blaser

M. Akdis

Date

Source

Serveur académique Lausannois (2)

Mot-clé

Diseases (2) Expression (2) Diseases (2) temps (durée) (1) acte législatif (1) analyse (1) allergie (1) différenciation (1) cancer (1) Differentiation (1) Development (1) Treatment (1) Cancer (1) Phenotype (1) Th2 cells (1) Transcription factors (1) Interleukin-4 (1) Transgenic mice (1) Neovascularization (1) Transcription (1) Toleration (1) Time (1) T cells (1) Process (Law) (1) Patients (1) Motion (1) Mice (1) Melanoma (1) Mechanics, Analytic (1) Human beings (1)

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A novel proangiogenic B cell subset is increased in cancer and chronic inflammation.

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GATA3-driven Th2 responses inhibit TGF-beta1-induced FOXP3 expression and the formation of regulatory T cells.

P.Y. Mantel et al. (2007)