L. Willen

Discipline

Histoire, Philosophie et Sociologie des sciences (3) Economies et finances (1)

Type de document

Articles (22)

Co-auteur

Date

Source

Mot-clé

Mice (13) Human beings (13) protéine (12) famille (11) Families (11) Receptors (11) Family (11) Cells (9) ligne (8) Development (8) Treatment (8) anticorps (7) survie (7) Tumor necrosis factor (7) Survival (7) Patients (7) Ligands (7) Immunoglobulins (7) B cells (7) Animals (7) Patients (7) Ectodermal dysplasia (6) Monoclonal antibodies (6) analyse (5) Inhibitors (5) Teeth (5) Inhibition (5) Oligomerization (5) Properties (5) administration (4)

Documents écrit par L. Willen (22) | parle de L. Willen (0)

Ligand-independent oligomerization of TACI is controlled by the transmembrane domain and regulates proliferation of activated B cells.
C.R. Smulski et al. (29 mars 2022)
Kinetics of free and ligand-bound atacicept in human serum.
M. Eslami et al. (2022)
APRIL limits atherosclerosis by binding to heparan sulfate proteoglycans.
D. Tsiantoulas et al. (25 août 2021)
BAFF 60-mer, and Differential BAFF 60-mer Dissociating Activities in Human Serum, Cord Blood and Cerebrospinal Fluid.
M. Eslami et al. (2020)
No interactions between heparin and atacicept, an antagonist of B cell survival cytokines.
C. Kowalczyk-Quintas et al.
A loop region of BAFF controls B cell survival and regulates recognition by different inhibitors.
M. Vigolo et al. (23 mars 2018)
Inhibition of Membrane-Bound BAFF by the Anti-BAFF Antibody Belimumab.
C. Kowalczyk-Quintas et al. (2018)
BAFF- and TACI-Dependent Processing of BAFFR by ADAM Proteases Regulates the Survival of B Cells.
C.R. Smulski et al. (28 févr. 2017)
Hetero-oligomerization between the TNF receptor superfamily members CD40, Fas and TRAILR2 modulate CD40 signalling.
C.R. Smulski et al. (9 févr. 2017)
Ectodysplasin A in Biological Fluids and Diagnosis of Ectodermal Dysplasia.
J. Podzus et al. (2017)
Antibodies That Block or Activate Mouse B Cell Activating Factor of the Tumor Necrosis Factor (TNF) Family (BAFF), Respectively, Induce B Cell Depletion or B Cell Hyperplasia.
C. Kowalczyk-Quintas et al. (16 sept. 2016)
Characterization and application of two RANK-specific antibodies with different biological activities.
M. Chypre et al. (2016)
Stoichiometry of heteromeric BAFF and APRIL cytokines dictates their receptor binding and signaling properties.
S. Schuepbach-Mallepell et al. (2015)
Pharmacological stimulation of edar signaling in the adult enhances sebaceous gland size and function.
C. Kowalczyk-Quintas et al. (2015)
Generation and characterization of function-blocking anti-ectodysplasin A (EDA) monoclonal antibodies that induce ectodermal dysplasia.
C. Kowalczyk-Quintas et al. (2014)
Human cytomegalovirus glycoprotein UL141 targets the TRAIL death receptors to thwart host innate antiviral defenses.
W. Smith et al. (2013)
No Evidence That Soluble TACI Induces Signalling via Membrane-Expressed BAFF and APRIL in Myeloid Cells.
J. Nys et al. (2013)
Structure of the extracellular domains of human and Xenopus Fn14: implications in the evolution of TWEAK and Fn14 interactions.
M. Pellegrini et al. (2013)
Molecular and therapeutic characterization of anti-ectodysplasin A receptor (EDAR) agonist monoclonal antibodies.
C. Kowalczyk et al. (2011)
Functional analysis of Ectodysplasin-A mutations causing selective tooth agenesis.
G. Mues et al. (2010)
Biological activity of ectodysplasin A is conditioned by its collagen and heparan sulfate proteoglycan-binding domains.
L.K. Swee et al.
TACI, unlike BAFF-R, is solely activated by oligomeric BAFF and APRIL to support survival of activated B cells and plasmablasts.
C. Bossen et al. (2008)