Tissu adipeux épicardique et fibrillation auriculaire [Epicardial adipose tissue and atrial fibrillation]

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25 mai 2022

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info:eu-repo/semantics/altIdentifier/doi/10.53738/REVMED.2022.18.783.1048

Ce document est lié à :
info:eu-repo/semantics/altIdentifier/pmid/35612477

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info:eu-repo/semantics/altIdentifier/pissn/1660-9379

Ce document est lié à :
info:eu-repo/semantics/altIdentifier/urn/urn:nbn:ch:serval-BIB_CC8D1A21E68A6

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info:eu-repo/semantics/openAccess , CC BY-NC-ND 4.0 , https://creativecommons.org/licenses/by-nc-nd/4.0/




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L. Iten et al., « Tissu adipeux épicardique et fibrillation auriculaire [Epicardial adipose tissue and atrial fibrillation] », Serveur académique Lausannois, ID : 10.53738/REVMED.2022.18.783.1048


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Atrial fibrillation (AF) is the most common arrhythmia encountered in adults; it is associated with a significant morbidity and mortality. Obesity is a risk factor contributing to AF occurrence. Recently, interest has focused on epicardial adipose tissue (EAT), defined as a fatty deposit located between the epicardium and the visceral pericardium. Its characteristics are distinct from classic adipose deposits: it infiltrates the epicardial myocardium and secretes cytokines, which modulate cardiomyocyte electrophysiology and cardiac remodeling. Different studies show that EAT can be an independent risk factor for AF and that EAT thickness, as measured by CT or MRI, could predict the presence, severity and recurrence of AF.

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