A.P. Bemelmans

Type de document

Articles (7)

Co-auteur

Date

Source

Mot-clé

Mice (7) protéine (6) Animals (6) spectacle (5) Retinal (Visual pigment) (5) modèle (4) perte (4) Treatment (4) Photoreceptors (4) Eye (4) Expression (4) Example (4) Cone (4) Cells (4) mutant (3) lumière (3) maladie (3) vecteur (3) Vision (3) Retinal degeneration (3) Retina (3) Human beings (3) Light (3) Genetic transformation (3) Gene therapy (3) Drama (3) Diseases--Animal models (3) Gene therapy (3) maladie (3) puits (2)

Documents écrit par A.P. Bemelmans (7) | parle de A.P. Bemelmans (0)

The striatal kinase DCLK3 produces neuroprotection against mutant huntingtin.
L. Galvan et al. (1 mai 2018)
Evaluation of tolerance to lentiviral LV-RPE65 gene therapy vector after subretinal delivery in non-human primates.
A. Matet et al.
Alzheimer's disease-like APP processing in wild-type mice identifies synaptic defects as initial steps of disease progression.
M. Audrain et al. (12 janv. 2016)
Hyperactivation of retina by light in mice leads to photoreceptor cell death mediated by VEGF and retinal pigment epithelium permeability.
M. Cachafeiro et al. (2013)
Gene therapy regenerates protein expression in cone photoreceptors in Rpe65(R91W/R91W) mice.
C. Kostic et al. (2011)
Remaining rod activity mediates visual behavior in adult Rpe65-/- mice.
M. Cachafeiro et al. (2010)
Lentiviral gene transfer of RPE65 rescues survival and function of cones in a mouse model of Leber congenital amaurosis.
A.P. Bemelmans et al. (2006)