C. Kostic

Discipline

Psychologie (4)

Type de document

Articles (24) Recensions (3) Colloques et conférences (1)

Co-auteur

Date

Source

Mot-clé

Mice (17) Retinal (Visual pigment) (14) Cells (12) Animals (12) Retinal degeneration (11) Eye (11) puits (10) gène (10) Cone (10) protéine (9) lumière (9) maladie (9) Retina (9) Light (9) Expression (9) maladie (9) spectacle (8) perte (8) Treatment (8) Photoreceptors (8) Gene therapy (8) vecteur (7) Development (7) Gene therapy (7) Example (7) Number (7) modèle (6) Vision (6) Human beings (6) Blindness (6)

Documents écrit par C. Kostic (24) | parle de C. Kostic (0)

Lentiviral Vectors for Ocular Gene Therapy.
Y. Arsenijevic et al. (31 juil. 2022)
The connecting cilium inner scaffold provides a structural foundation that protects against retinal degeneration.
O. Mercey et al.
Enhancer of Zeste Homolog 2 (EZH2) Contributes to Rod Photoreceptor Death Process in Several Forms of Retinal Degeneration and Its Activity Can Serve as a Biomarker for Therapy Efficacy.
M. Mbefo et al. (28 août 2021)
Quantification of the early pupillary dilation kinetic to assess rod and cone activity.
C. Kostic et al. (5 mai 2021)
Lentiviral mediated RPE65 gene transfer in healthy hiPSCs-derived retinal pigment epithelial cells markedly increased RPE65 mRNA, but modestly protein level.
F. Udry et al. (1 juin 2020)
Maturation of the Pupil Light Reflex Occurs Until Adulthood in Mice.
N. Kircher et al. (2019)
Evaluation of tolerance to lentiviral LV-RPE65 gene therapy vector after subretinal delivery in non-human primates.
A. Matet et al.
Rai1 frees mice from the repression of active wake behaviors by light.
S. Diessler et al. (2017)
Adamts18 deletion results in distinct developmental defects and provides a model for congenital disorders of lens, lung, and female reproductive tract development.
D. Ataca et al. (15 nov. 2016)
Amyloid Precursor-Like Protein 2 deletion-induced retinal synaptopathy related to congenital stationary night blindness: structural, functional and molecular characteristics.
V. Dinet et al. (8 juin 2016)
Determination of Rod and Cone Influence to the Early and Late Dynamic of the Pupillary Light Response.
C. Kostic et al.
Animal modelling for inherited central vision loss.
C. Kostic et al. (2016)
Multigenic lentiviral vectors for combined and tissue-specific expression of miRNA- and protein-based antiangiogenic factors.
A.L. Askou et al. (2015)
Hyperactivation of retina by light in mice leads to photoreceptor cell death mediated by VEGF and retinal pigment epithelium permeability.
M. Cachafeiro et al. (2013)
Rapid cohort generation and analysis of disease spectrum of large animal model of cone dystrophy.
C. Kostic et al. (2013)
FAM161A, associated with retinitis pigmentosa, is a component of the cilia-basal body complex and interacts with proteins involved in ciliopathies.
S.A. Di Gioia et al. (2012)
Retinal degeneration progression changes lentiviral vector cell targeting in the retina.
M. Calame et al. (2011)
Gene therapy regenerates protein expression in cone photoreceptors in Rpe65(R91W/R91W) mice.
C. Kostic et al. (2011)
Remaining rod activity mediates visual behavior in adult Rpe65-/- mice.
M. Cachafeiro et al. (2010)
In conditions of limited chromophore supply rods entrap 11-cis-retinal leading to loss of cone function and cell death.
M. Samardzija et al. (2009)
Ophtalmologie. Thérapie génique des rétinopathies héréditaires: premiers résultats [Gene therapy for hereditary eye diseases: where are we?]
H. Viet Tran et al. (2009)
When green algae give light to blind mice
Y. Arsenijevic et al. (2008)
Lentiviral gene transfer of RPE65 rescues survival and function of cones in a mouse model of Leber congenital amaurosis.
A.P. Bemelmans et al. (2006)
Bmi1 loss produces an increase in astroglial cells and a decrease in neural stem cell population and proliferation
D. Zencak et al.