J.H. Cabungcal

Discipline

Psychologie (8)

Type de document

Articles (19) Recensions (2)

Co-auteur

Date

Source

Mot-clé

Schizophrenia (16) Oxidative stress (15) Mice (12) Interneurons (11) Patients (10) Example (10) Animals (10) Patients (10) modèle (9) Human beings (9) Treatment (8) adulte (7) Development (7) Nets (7) Integrity (7) Brain (7) adulte (7) cerveau (7) Rats (6) Physiology, Pathological (6) Neurons (6) Pathophysiology (6) spectacle (5) Control (5) Prefrontal cortex (5) Evidence (5) Diseases--Animal models (5) Disabilities (5) puits (4) thérapie (4)

Documents écrit par J.H. Cabungcal (19) | parle de J.H. Cabungcal (0)

Low protein-induced intrauterine growth restriction as a risk factor for schizophrenia phenotype in a rat model: assessing the role of oxidative stress and neuroinflammation interaction.
L. Allgäuer et al. (1 févr. 2023)
Caught in vicious circles: a perspective on dynamic feed-forward loops driving oxidative stress in schizophrenia.
M. Cuenod et al.
Developmental oxidative stress leads to T-type Ca2+ channel hypofunction in thalamic reticular nucleus of mouse models pertinent to schizophrenia.
C. El Khoueiry et al.
Mitochondrial, exosomal miR137-COX6A2 and gamma synchrony as biomarkers of parvalbumin interneurons, psychopathology, and neurocognition in schizophrenia.
I. Khadimallah et al.
Thalamic reticular nucleus impairments and abnormal prefrontal control of dopamine system in a developmental model of schizophrenia: prevention by N-acetylcysteine.
X. Zhu et al.
Timely N-Acetyl-Cysteine and Environmental Enrichment Rescue Oxidative Stress-Induced Parvalbumin Interneuron Impairments via MMP9/RAGE Pathway: A Translational Approach for Early Intervention in Psychosis.
D. Dwir et al. (21 oct. 2021)
MMP9/RAGE pathway overactivation mediates redox dysregulation and neuroinflammation, leading to inhibitory/excitatory imbalance: a reverse translation study in schizophrenia patients.
D. Dwir et al.
S.11.03 Similar anomalies in the thalamic reticular nucleus of a mice model of redox dysregulation and of schizophrenia patients
P. Steullet et al. (2019)
Nutritional Intervention for Developmental Brain Damage: Effects of Lactoferrin Supplementation in Hypocaloric Induced Intrauterine Growth Restriction Rat Pups.
Y. van de Looij et al. (2019)
The thalamic reticular nucleus in schizophrenia and bipolar disorder: role of parvalbumin-expressing neuron networks and oxidative stress.
P. Steullet et al.
A lack of GluN2A-containing NMDA receptors confers a vulnerability to redox dysregulation: Consequences on parvalbumin interneurons, and their perineuronal nets.
R. Cardis et al.
Nrf2-dependent persistent oxidative stress results in stress-induced vulnerability to depression.
E. Bouvier et al.
Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia.
P. Steullet et al.
Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?
P. Steullet et al.
Glutathione deficit impairs myelin maturation: relevance for white matter integrity in schizophrenia patients.
A. Monin et al.
Cortical fast-spiking parvalbumin interneurons enwrapped in the perineuronal net express the metallopeptidases Adamts8, Adamts15 and Neprilysin.
J. Rossier et al. (2015)
Juvenile antioxidant treatment prevents adult deficits in a developmental model of schizophrenia.
J.H. Cabungcal et al. (2014)
Fast oscillatory activity in the anterior cingulate cortex: dopaminergic modulation and effect of perineuronal net loss.
P. Steullet et al. (2014)
Redox dysregulation affects the ventral but not dorsal hippocampus: impairment of parvalbumin neurons, gamma oscillations, and related behaviors.
P. Steullet et al. (2010)