P. Steullet

Discipline

Psychologie (6)

Type de document

Articles (17) Recensions (3)

Co-auteur

Date

Source

Mot-clé

Schizophrenia (16) Mice (13) Oxidative stress (12) Patients (11) Patients (11) Human beings (9) Animals (9) Interneurons (8) adulte (7) Development (7) Integrity (7) adulte (7) modèle (6) Psychoses (6) Physiology, Pathological (6) Example (6) Brain (6) Pathophysiology (6) cerveau (6) oxydoréduction (5) Treatment (5) Neurons (5) Glutathione (5) Synthesis (5) maladie (5) spectacle (4) maladie (4) Prefrontal cortex (4) Oscillations (4) Nets (4)

Documents écrit par P. Steullet (17) | parle de P. Steullet (0)

Time of exposure to social defeat stress during childhood and adolescence and redox dysregulation on long-lasting behavioral changes, a translational study.
M. Schnider et al. (26 sept. 2022)
Caught in vicious circles: a perspective on dynamic feed-forward loops driving oxidative stress in schizophrenia.
M. Cuenod et al.
Developmental oxidative stress leads to T-type Ca2+ channel hypofunction in thalamic reticular nucleus of mouse models pertinent to schizophrenia.
C. El Khoueiry et al.
Mitochondrial, exosomal miR137-COX6A2 and gamma synchrony as biomarkers of parvalbumin interneurons, psychopathology, and neurocognition in schizophrenia.
I. Khadimallah et al.
Timely N-Acetyl-Cysteine and Environmental Enrichment Rescue Oxidative Stress-Induced Parvalbumin Interneuron Impairments via MMP9/RAGE Pathway: A Translational Approach for Early Intervention in Psychosis.
D. Dwir et al. (21 oct. 2021)
Thalamus-related anomalies as candidate mechanism-based biomarkers for psychosis.
P. Steullet
MMP9/RAGE pathway overactivation mediates redox dysregulation and neuroinflammation, leading to inhibitory/excitatory imbalance: a reverse translation study in schizophrenia patients.
D. Dwir et al.
S.11.03 Similar anomalies in the thalamic reticular nucleus of a mice model of redox dysregulation and of schizophrenia patients
P. Steullet et al. (2019)
The thalamic reticular nucleus in schizophrenia and bipolar disorder: role of parvalbumin-expressing neuron networks and oxidative stress.
P. Steullet et al.
A lack of GluN2A-containing NMDA receptors confers a vulnerability to redox dysregulation: Consequences on parvalbumin interneurons, and their perineuronal nets.
R. Cardis et al.
Oxidative stress-driven parvalbumin interneuron impairment as a common mechanism in models of schizophrenia.
P. Steullet et al.
Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?
P. Steullet et al.
Glutamate Cysteine Ligase-Modulatory Subunit Knockout Mouse Shows Normal Insulin Sensitivity but Reduced Liver Glycogen Storage.
S. Lavoie et al. (2016)
Glutathione Deficit Affects the Integrity and Function of the Fimbria/Fornix and Anterior Commissure in Mice: Relevance for Schizophrenia.
A. Corcoba et al. (3 oct. 2015)
Glutathione deficit impairs myelin maturation: relevance for white matter integrity in schizophrenia patients.
A. Monin et al.
Fast oscillatory activity in the anterior cingulate cortex: dopaminergic modulation and effect of perineuronal net loss.
P. Steullet et al. (2014)
Redox dysregulation affects the ventral but not dorsal hippocampus: impairment of parvalbumin neurons, gamma oscillations, and related behaviors.
P. Steullet et al. (2010)