D. Dwir

Discipline

Psychologie (2)

Type de document

Articles (8) Recensions (2)

Co-auteur

Date

Source

Mot-clé

Schizophrenia (7) Oxidative stress (7) Patients (6) Patients (6) Development (4) Interneurons (4) Physiology, Pathological (4) Mice (4) Human beings (4) Integrity (4) Evidence (4) Pathophysiology (4) risque (3) recherche (3) oxydoréduction (3) gène (3) risque pour l'environnement (3) adulte (3) abords (agglomération) (3) Risk (3) Research (3) Psychiatry (3) Nets (3) Microglia (3) Manufacturers' agents (3) Inflammation (3) Expression (3) Commission merchants (3) Brain (3) Biochemical markers (3)

Documents écrit par D. Dwir (8) | parle de D. Dwir (0)

Characterization of early psychosis patients carrying a genetic vulnerability to redox dysregulation: a computational analysis of mechanism-based gene expression profile in fibroblasts.
B. Giangreco et al.
Low protein-induced intrauterine growth restriction as a risk factor for schizophrenia phenotype in a rat model: assessing the role of oxidative stress and neuroinflammation interaction.
L. Allgäuer et al. (1 févr. 2023)
Caught in vicious circles: a perspective on dynamic feed-forward loops driving oxidative stress in schizophrenia.
M. Cuenod et al.
Timely N-Acetyl-Cysteine and Environmental Enrichment Rescue Oxidative Stress-Induced Parvalbumin Interneuron Impairments via MMP9/RAGE Pathway: A Translational Approach for Early Intervention in Psychosis.
D. Dwir et al. (21 oct. 2021)
MMP9/RAGE pathway overactivation mediates redox dysregulation and neuroinflammation, leading to inhibitory/excitatory imbalance: a reverse translation study in schizophrenia patients.
D. Dwir et al.
A lack of GluN2A-containing NMDA receptors confers a vulnerability to redox dysregulation: Consequences on parvalbumin interneurons, and their perineuronal nets.
R. Cardis et al.
Tollip, an early regulator of the acute inflammatory response in the substantia nigra.
M. Humbert-Claude et al. (7 déc. 2016)
Redox dysregulation, neuroinflammation, and NMDA receptor hypofunction: A "central hub" in schizophrenia pathophysiology?
P. Steullet et al.